Mechanism of Action
Investigative studies conducted overseas have shown that terlipressin, the active agent in BIV-201, has the potential to reduce the presence of ascites in the abdomen through vasoconstricting the blood vessels where the blood is pooling, hence restoring blood flow through the kidneys and liver. This reduces portal vein pressure and increases blood volume in the arteries. Consequently, the body may respond by shutting down the RAAS system which has been generating the ascites.
Terlipressin has been available for decades outside the US, but has not been available in the US or Japan. It is approved in more than 40 countries for in hospital use for the treatment of two deadly conditions related to ascites called bleeding esophageal varices (BEV) and hepatorenal syndrome (HRS). The drug has been studied extensively overseas.